RECENT STUDIES SHOW THAT UP TO 10% OF LYME INFECTED INDIVIDUALS IN THE MIDWEST MAY BE CARRIERS OF POWASSAN VIRUS: Powassan Virus has been an emerging cause of neuroinvasive disease in the upper Midwest. The following studies describe the prevalence and geographic distribution of ticks carrying Powassan Virus as well as Borrelia Burgdorferi (the causative agent of Lyme disease.) These findings suggest that concurrent transmission of Powassan Virus and Borrelia Burgdorferi from coinfected ticks is likely to occur in humans. In a recent study done by the Medical College of Wisconsin, Powassan Virus is described to be present in around 10% of tickborne disease patients -- 1/3 of which tested positive for IgM antibodies. Thus, it can be concluded that in a Lyme disease endemic area, Powassan Virus should be considered to be a possible diagnosis for patients suffering from brain encephalitic symptoms and/or for cases in which Lyme treatments have failed. The spectrum of disease is broader than previously realized with most patients having mild symptomatic infection. Symptoms may include: occasional confusion; slurred speech; joint pain; fatigue; chills and sweats; headaches; head pressure; reduced motor functions; nausea; weakness. In more severe cases, encephalitis that could extend to meningoencephalitis may occur, which is a condition that simultaneously affects the brain and the spinal cord. Treatment options for Powassan Virus are limited; however, we have seen great improvement in patients who have received hyperthermia. Refractory cases have required a more extensive approach. #lymemexico#dromarmorales#powassanvirus#lymeawareness
Aparecida, tem vários fatores que leva à cefaleia. Porém a localização da dor é importante para um avaliação clínica. Pela razão di chiado do ouvido, à princípio procure um Otorrinolaringologista. Ele dirá a necessidade de vc procurar um Neuro para possíveis exames de imagens computadorizados. O uso de paracetamol poderá mascará um problema sério. Abraço.
Lyme disease was made inside in the lab. Ticks were found to be the best carriers for it.
Eu tenho dor com pressão na cabeça , só de um lado,
e chiado no ouvido DIRETO , tomo paracetamol 🤷🤷🤷
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Dr. Morales’ Thoughts, Part 2: Immunoablation and autologous hematopoietic stem cell transplantation (IAHSCT)” It is a technique used for resetting the immune system, replacing a diseased immune system with a new one, providing a long-term remission. There is now emerging evidence that immunoablation followed by ASCT provides fundamental changes of the immune system that may rewire and manipulate a chronic autoimmune system into a naïve and self-tolerant state. IAHSCT has proven effective in extreme stubborn cases where autoimmunity, regardless of its origin, has been resistant to steroid and immune suppressive therapy. Trigger factors for autoimmunity may include: infection; vaccines; environmental or chemical exposure; DNA disruption; injury or cellular stress. The immune system is thus left in an overreactive state, producing damage against itself, attacking its own cells and organs, such as multiple endocrine glands, neuronal tissue, optic nerves, gallbladder, lungs, skin, digestive system, CNS & even blood components. IAHSCT could potentially help patients suffering from persistent autoimmune disorders triggered by chronic tick-borne infections, other vector transmitted illnesses, neurodegenerative diseases i.e. ALS, MS and other autoimmune mediated disorders refractory to treatment. IAHSCT is a two-step procedure: The first step begins by giving a person a medication that moves their hematopoietic stem cells from the bone marrow into their blood. These stem cells are then collected from the blood by apheresis, purified and prepared. Stem cells can also be harvested by direct bone marrow aspiration. The second step begins with chemotherapy drugs which are used to suppress the person’s diseased immune system. The stem cells collected in the first step are transplanted back into the same person, to give rise to an enhanced immune system. There are many potential side effects from the chemotherapy, and patients require specialized selection and care. www.clinexprheumatol.org/article.asp?a=10846#lymemexico#dromarmorales#lymedisease... See MoreSee Less
Dr. Morales’ Thoughts, Part 1: Over the recent past years, researchers have sought to describe the various mechanisms of Lyme disease that act by suppressing the human’s innate immune response. According to such findings, the immune system is rendered inert or unable to recognize the presence of bacteria or becomes corrupted entirely. It is also believed that the immune system reacts to bacterial liposomal vesicles adhered to nerve tissues, attacking them and mistakenly attacking multiple neuronal tissues as well. The onset of chronic symptoms thereafter is sometimes mistakenly attributed to an autoimmune disease. Taken to its logical conclusion, some misdiagnosed autoimmune diseases may be a direct result of the Lyme infection itself. These bacterial mechanisms mostly consist of outer surface proteins and plasmids that are able to attach firmly to lymphocytic receptor binding sites, making them less sensitive to or aware of the presence of Lyme bacteria and, in some cases, becoming overreactive against its own host. Consequently, this causes both a primary immune deficiency along with a contemporaneous autoimmune response. As evolving studies suggest that individuals without any genetic predisposition can develop inflammation and autoimmunity triggered from bacterial infection, some cases where Lyme has been successfully managed, may show signs of persistence of such autoimmunity. Supported by our local resources, we have been able to manipulate T lymphocytes both in vitro and in vivo by means of autologous Dendritic cells extracted by apheresis. This allows the correction of the stubborn autoimmune response and suppression of auto-antibody expression by re-introducing tolerance through T-reg cells derived from Dendritic cell interactions. However, as favorable outcomes have been observed by the use of this technique, some extreme cases displaying higher predisposition for autoimmunity may require immune ablation and hematopoietic stem cell transplant (HSCT) to correct its path. By Dr. Omar Morales For more information about this or other mechanism of persistence and chronicity, please write to us at firstname.lastname@example.org. #lymedisease#lymemexico... See MoreSee Less
Para o mundo inteiro saber e ver. Viva México.
What about small fiber neuropathy, what can be done?
All Borrellia species attack differently and have varying antigens some may attack the neck others the lower back some multiple areas. How is your approach do you have a mapped out system and how do yo treat bartonella so up regulate the immune system
the only truth about lyme www.truthcures.org
Me explica em resumo.... José Maria Soares Feitosa.
El misterio de la ciencia
DR. DAVID JAMES: CHEMTRAILS CAUSA ENFERMEDAD DE LYMES
El Dr. David James habla sobre la correlación de la enfermedad de Lyme y las estelas químicas. Si la atmósfera está siendo manipulada como lo es ahora a través de mutaciones y nano contaminantes, la química cambia.
Nuestro sistema inmunitario no reconoce las toxinas en el aire junto con lo que se rocía sobre nosotros, y no reacciona para eliminar estas toxinas. La pulverización de óxido de alúmina, óxido de bario, micoplasmas, virus, bacterias.
Cuando estos patógenos interactúan, algunos mueren para crear una biopelícula, que luego se une entre sí. Dado que estas partículas se utilizan para producir en masa, esto también sucedería a través de los cócteles químicos en la atmósfera. El calor a través de la tecnología HAARP cargaría las frecuencias y crearía una mutación. La bacteria se transformaría en algo que no estaba destinado; un estofado tóxico químico / patógeno.
Todo lo que sube tiene que bajar. Una atmósfera genéticamente modificada que estamos comiendo, respirando y bebiendo.